"I am probably learning more than he is by reading your posts"
There actually isn't anything in his posts that I have not heard before. Thankfully, since I restrict myself to evolutionary materials to teach me about evolution, I don't tend to be surprised. Much of the post is getting off track, and as much as I hate leaving things unanswered, I'm moving on.
With one exception, because obviously it goes to my own hypothesis, which is what I'm using to unhinge myself from requiring a certain amount of 'time' before I tend to the actual question of ascertaining exactly how much time there is.
I would be interested in hearing your source, Broken, for your assertion that the human genome is composed of 30% transposable elements. I am quite certain that you are over-stating it, but as evolutionists are constantly changing their mind, perhaps the consensus has changed. There is one critical reason why TE's do not help you:
No selectively valuable contributions are known for ANY of the TEs. Rather they often seem deleterious, but natural selection seems unable to eliminate them (which by God, you'd think it could).
Since they don't typically have effects that can be selected for, you gain nothing by proposing them. When they do, they are deleterious. Also, when they happen they may cause mutations on an adjacent gene, which while 'new information,' given the fact that most mutations are deleterious, that's not saying much.
[edited because I found Broken said something that confirms the above]
You yourself said:
"When a TE inserts itself into a gene's regulatory region (their usual target), the gene's function is alterred,
mostly for the worse, but sometimes for the better."
Now, let's compare that to what I said that started this line of discourse:
My POV is simply that you are invoking observed exceptions to the observed rules we observe in order to create an imagined rule into the unfalsifiable past. You are welcome to do that, if you like. I consider that epistemologically weak, and a lot of folks would agree with me.
So, you admit that when a TE inserts itself, though it affects gene function, it is MOSTLY FOR THE WORSE. Let's compare that with what I said....
YOU ARE INVOKING EXCEPTIONS TO THE OBSERVED RULES. What are the observed rules? You said it yourself: MOSTLY FOR THE WORSE.
Perhaps my English is poor, but 'MOSTLY' seems consistent with 'OBSERVED RULES,' while 'SOMETIMES' would seem to be consistent with EXCEPTIONS.
In otherwords, the point that I am actually making you yourself confirm virtually explicitly. Your problem consists mainly in not recognizing my point, but also because you are comfortable to create rules out of exceptions and exceptions out of the rules.
Whose hypothesis does TE's support? Mine. Which hypothesis does it provide EMPIRICAL support for? Mine.
To that end, I thank you for mentioning them.
[/end edit]
You also fail to understand your own theory. At heart, EVERY bit of the gene code must, by definition, have been a 'mutation' at one time. The presumption is that these mutations have been sifted through resulting in productive organisms. So, telling me that we observe 'new information' coming in when in principle EVERY bit of ANY genome necessarily was 'new' at one time is.... well... uninteresting and unimpressive.
Now hang on a second while I do a quick look in my biology text book to see if it has anything more d--ning than what I just said...
I quote:
"What role do these repeated sequences play in the cell? There are few answers."
lol. That's ok, Broken. You just keep trotting TE's out. Here follows a handful of statements about 'possibilities.' Now, perhaps the most interesting thing about the TEs in my textbook (besides the fact that they are mainly only talked about having any real manifestations in regards to BACTERIA, is the fact that they are only talked about on 5 pages, and then not very much. The included teh comment above "There are few answers." lol
A text book that has 1241 pages and is obnoxiously evolutionary in nature has only 5 pages containing any discussion on TEs, and then, in the main, expresses uncertainty about their 'role.' They get a little bit better play in regards to bacteria.
And it may be interesting to further note that for something you are apparently quite confident in parading out as the coup de grace, Chapter 20 "The Mechanisms of Evolution" doesn't bother to mention them at all.
Maybe they are not as important as you think they are. Perhaps you should consult the editors and tell them that they are forgetting to mention some of the best evidence for evolution!
This is a 1998 book. Evolutionists, as I said, are constantly changing their mind, so perhaps views have changed. I stand by my assessment above that "No selectively valuable contributions are known for ANY of the TEs. Rather they often seem deleterious, but natural selection seems unable to eliminate them (which by God, you'd think it could)."
That sums up what I knew based on information I picked up before I bought and began to read the text book.
Oh yes, my friends. Virtually every night before I go to bed I read the textbooks and primary sources that are produced by my opposition. And you thought I read my Bible.

It saddens me how rarely I need to speak to specifics. Its practically a waste of time, since most arguments are around generalities. In that respect, then, I thank you Broken, for allowing me to trot out some of my latent reserves of otherwise worthless knowledge.
I especially thank you for trotting out what seems to be a practically worthless feature in support of your position. If I were an evolutionist trying to support my position, I'd leave the TE's out. And it would be nice if some of my opponents spent as much time reading the primary sources and texts produced by MY side (not merely speaking of YEC, here). We'd be able to cut through a lot of the bullcrap.
Allow me to summarize: The General Rule is that there is variation with limits. That is the observation.
TEs do not produce noticeable variation. They operate almost parasitically. As such, even they do not speak to the question of 'variation with limits,' but to the extent that they speak to anything, they support my hypothesis, which maintains not that every part of the genome was at one time in history a 'mutant', but that the genome in a given 'class' was itself 'perfectly complete.'
Hey, you've heard the saying,